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To distinguish between a specific versus a random humoral response, we sequenced IgG heavy (V/H) and light (V/L) chain variable regions expressed in multiple acute MS plaques and found a restricted response consisting primarily of V/H4 germline genes. Some V/H sequences were over-represented, clonally expanded, and displayed a non-random accumulation of somatic mutations, features indicative of an antigen driven B cell response. A parallel analysis of subacute sclerosing panencephalitis (SSPE) brain also revealed over-represented and somatically mutated V/H and V/L sequences.

Emerging research on the molecular etiology and pathogenesis of disease suggests that genetic diseases can be further partitioned into those that are heritable and those thought to arise from the accumulation of acquired genetic damage. This award is 26 Sclerosis designed to permit the PI to permanently leave his position as a radiation biologist, move into research and teaching in the field of aging full-time, receive training in subjects relevant to biodemographic research, and pursue a series of research projects associated with the training that initially focus on the biodemography of genetic diseases in humans.

Neurologic deficits and spinal cord C-tau levels will be determined in ALS and age-matched control mice and statistically compared from the presymptomatic stages to late symptomatic stages of disease progression. Specific Aim 3: Determine if C-tau levels reliably quantify the effect of a demonstrated neuroprotectant drag intervention in ALS mice. C-tau levels and rotorod performance will be determined and compared as a function of NDGA treatment. Generate_Screen • Project Title: BONE MARROW DERIVED MESANGIAL CELL PROGENITORS IN AGING Principal Investigator & Institution: Striker, Gary E.

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